in hypovolemic shock, there is a deficiency in blood cells (hence the name, Hypo.Vol.Emic) thus the blood vessels react to this by contracting in hopes of increasing cardiac preload by increasing blood pressure (So Blood Vessels contract > blood pressure increases > preload increases.) but how does this lead to an increased preload? Isn’t the heart just receiving the same quantity of blood but throughout a longer period? (So small amounts throughout a longer period instead of a big amount throughout a short period)
I think I’ve got this whole thing tangled up
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Turns a low pressure low volume system into a high pressure high volume system. Delivering more blood (preload) therefore hopefully (if EF preserved) increased cardiac output etc.
I agree it’s a bit tough sometimes to get it right in the head. Brings back days as a cardiothoracic jnr Dr.
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