in hypovolemic shock, there is a deficiency in blood cells (hence the name, Hypo.Vol.Emic) thus the blood vessels react to this by contracting in hopes of increasing cardiac preload by increasing blood pressure (So Blood Vessels contract > blood pressure increases > preload increases.) but how does this lead to an increased preload? Isn’t the heart just receiving the same quantity of blood but throughout a longer period? (So small amounts throughout a longer period instead of a big amount throughout a short period)
I think I’ve got this whole thing tangled up
In: 1
Most of your total vasculature is in the peripheries / not in the great vessels.
Widespread vasoconstriction therefore preferentially forces blood away from the peripheries and towards the heart.
This results in increased preload.
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