Pain receptors emit the greatest signal just before tissue is actually injured or destroyed. Among the stimuli receptors respond to is tension within tissue. Once the injury occurs, tension is released and the pain subsides. Friction and probing of already injured tissue produces less pain than the original injury.
Once the shunt is stabilized, it no longer moves, so it stimulates no additional pain receptors. Receptors that originally signalled injury settle into their new home, recover, and stop emitting.
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