I’m going off memory here:
Viruses need something to latch on to, receptors on cells, before it can do what viruses do , infect cells, replicate, and spread.
One of the differences that SARS-CoV-2 has over -1 is that the receptors that it uses are more plentiful, found all over the respiratory system, and else where, instead of -1 which was primary in the lungs.
If I recall correctly, this fact also led to there being less asymptomatic (cases without symptoms) cases with -1 so it was somewhat easier to track and control.
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